Otogenic hydrocephalus

[Summary]
Affected sinus thrombosis phlebitis, and obstacles arising from cerebral venous circulation, increased intracranial pressure for performance. Which is characterized by a bulging cerebral venous stasis, and edema. But high pressure brain ventricle expansion, and the general increase in ventricular cerebrospinal fluid expansion hydrocephalus different, it said Otogenic hydrocephalus, also known as Otogenic intracranial hypertension. Otogenic there are two types of hydrocephalus, cerebrospinal fluid increased ventricle first true expansion of hydrocephalus, first side sinus thrombosis in the aftermath of phlebitis circulatory disturbance of cerebral venous intracranial hypertension, that is, pseudo hydrocephalus. The latter is more多见.
[Diagnosis]
With the recent intracranial complications such as sinus thrombosis side phlebitis, lumbar puncture high pressure, and sometimes retinal edema, without occupying signs, and mri can be excluded ct intracranial space-occupying lesions. Superior sagittal sinus venous angiography showed a lateral sinus thrombosis.
[Treatment]
Treatment for dehydration, such as glycerol oral, or 250 ml of 20% mannitol, 2 to 3 times daily intravenous drip. Day or every other day for lumbar puncture, released 30 ~ 50 ml of CSF. If sexual edema and retinal optic nerve atrophy, to save vision, should be promptly adopted under the temporalis muscle or decompression of the lateral ventricle drainage. If the true hydrocephalus disease, should be lateral ventricle external jugular vein anastomosis, or thoracic spinal canal anastomosis.
[Etiology:
1. True hydrocephalus intracranial infection with meningitis and brain abscess, the result of adhesion or arachnoid aqueduct obstruction of cerebrospinal fluid caused increased secretion or absorption decline, resulting in increased cerebrospinal fluid form of hydrocephalus.
2. False hydrocephalus side sinus thrombosis phlebitis caused the right-side or both sides of sinus side at the same time sinus thrombosis, venous stasis caused brain edema, hemorrhage cortex point, the softening of the brain tissue infarction and cerebral pressure suddenly, but CSF small ventricle is not expansion, but is smaller compression phenomenon, and are thus false hydrocephalus. Side sinus thrombosis with phlebitis rate of 2.5%.
[Clinical]
Persistent headache, nausea, visual sexual weakened, or even totally blind, and this can sometimes accompanied by eye muscle paralysis, and other outreach.