Saturday, March 29, 2008

Why easy allergic rhinitis with secretory otitis media?

From anatomical structure, Eustachian tube through the nose and ears in contact. Eustachian tube in the mouth pharyngeal wall of the nasopharynx, from the inferior turbinate back about 1 ~ 1.5 cm. It inferior turbinate swelling or hypertrophy often cause nasal obstruction, ventilation and drainage tube impact arising from tinnitus and hearing loss and other ear symptoms.
The incidence of allergic rhinitis is a type I allergic. The original body after inhalation of allergen, the IgE in the nasal mucosa with shallow and superficial mast cells, eosinophils cell membrane, the nasal mucosa are sensitized to the state. When the change should be re-entering the original material nasal mucosa, the changes should be original and mast cells, eosinophils surface IgE occurred bridged, and then inspired a series of biochemical reactions that lead to a variety of media-based histamine release. These media through their respective vessel wall in the nasal mucosa, glands and nerve endings on the receptor, small blood vessel dilation, increased vascular permeability, exudative increase in inflammatory cell infiltration, edema, and so enhance the excitability of nerve endings . It nasal obvious edema, and affected the eustachian tube ventilation and drainage, and nasal, nasopharyngeal mucosa and the eustachian tube epithelial structure similar. Allergic impact on the same subject, both in its mucosal edema or effusion state can be caused by the narrow and the eustachian tube obstruction to bad ventilation function, as a result, a series of secretory otitis media performance such as bulging ear fullness ear, hearing loss, attic effusion, and so on.
Under normal circumstances, the Eustachian tube with adjustment mechanism will enable the middle ear cavity pressure and maintain a balance outside atmospheric pressure. When attic air gradually being absorbed, the eustachian tube in swallowing or when instantaneous open so that the nasal cavity from the Clean Air Temperature and Humidity and nasopharyngeal and oropharyngeal take advantage of temporary separation into the middle ear cavity, so that the dark room air can be added, while also draining role of the middle ear through the eustachian tube mucosal secretions can cilia movement since from the nasopharynx to the middle ear. When nasal allergic rhinitis caused edema, the eustachian tube is blocked, middle ear mucosa of air gradually being absorbed, and timely access to the outside world gas attic, the dark indoor formed negative pressure, can cause middle ear mucosa vascular expansion, Stasis blood, the vascular wall permeability increase in leakage of fluid attic. If the suction is not lifted, middle ear mucosal epithelial thickening can occur, the cell metaplasia, goblet cells increased secretions increase, thereby triggering secretory otitis media.
Therefore, allergic rhinitis easily with secretory otitis media.

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