What are the acute glomerulonephritis complications?

Severe acute phase of the major complications of a serious cycle of congestive state, hypertensive encephalopathy and acute renal failure. As in recent years to strengthen its work on the prevention of incidence and mortality rate has decreased significantly. 1. Cycle congestive state: because , blood volume dried up, until pulmonary edema. The incidence of various different reports, With severity, treatment-related. China 50 to 60 years reported to be hospitalized children with acute nephritis of 24% ~ 27% in seeing such complications, in recent years, the report has dropped to 2.4%. Many occur in the onset of acute nephritis after 1 to 2 weeks. Clinical manifestations of short breath, not supine, chest tightness, cough, lung wet-end audio, liver great tenderness,law about heart failure and other symptoms caused by the result of blood volume expansion, and true myocardial pump exhaust different. Often at this time of increased cardiac out and not reduced, the normal cycle time, moving no increase blood oxygen partial pressure difference, and digitalis category tonic effect poor, and often the application of diuretics to ease. A handful of severe development to be truly heart failure, in a few hours to 1 to 2 days and the rapidly emerging life-threatening pulmonary edema. 2. Hypertensive encephalopathy: mean blood pressure (DBP in particular) dramatically increased, in terms of a central nervous symptoms. Common children than adults in general. Usually that TB is on the basis of systemic hypertension, cerebral vasospasm resistance small brain edema caused cerebral hypoxia caused, but also some people believe that the rapid rise of blood pressure, cerebral vascular relaxation and contraction of a regulatory function of the automatic control, highly cerebral vascular congestion, a result of cerebral edema, acute nephritis Besides the also play a role in the pathogenesis. Many occurred in the early course of acute nephritis, a general acute onset showed severe headache, frequent nausea and vomiting, followed visual impairment, vertigo, diplopia, temporary and Mongolia, and lethargy or irritability, if not promptly treatment occurred convulsions, coma, a few temporary hemiplegia aphasia, when severe cerebral hernia. No more nerve of limitations signs, and shallow reflection tendon reflexes can weaken or disappear, ankle clonus sometimes positive, but also a pathological reflex, can have serious signs and symptoms of cerebral hernia. Fundus examination common retinal arteries spasm, sometimes visible papilledema. CSF Qingliang, pressure and protein in normal or increase slightly. If the blood pressure exceeds 18.7/12.0 kPa (140/90mmHg), with a visual impairment, convulsions and coma can be a diagnosis of the three. 3. Acute renal failure: a significant portion of children with acute nephritis in the acute phase with varying degrees of azotemia, and the progress for acute renal failure were only a handful. Complications still lack effective preventive measures, acute nephritis has become the leading cause of death. Clinical manifestations of oliguria or no urine, blood urea nitrogen, serum creatinine increased, hyperkalemia, metabolic acidosis. Oliguria or no urine for three to five or one week more, then urine volume increased symptoms disappeared, kidney function gradually resume. 4, secondary bacterial infection, acute nephritis as lower body resistance and easy to secondary infection, the most common is the lungs and urinary tract infection, in case of a secondary infection, they should actively symptomatic treatment to prevent the original disease heavier.