Cestan - Chenais's syndrome

Overview said, Cestan's syndrome type II. Cause pathogenesis of this levy paralysis found in pontine lesions located in the lateral part of the medulla oblongata. Caused by cerebral artery embolization of the larger branches caused occasionally tumor, infection, disease, medullary empty. Clinical manifestations of ipsilateral lesion soft palate and vocal cord paralysis, dysphagia and dysarthria, dizziness, vomiting, nystagmus, ipsilateral to the dumping, ataxia, facial sensory dysfunction, Horner's syndrome; contralateral lesions pain and temperature sense obstacles and pyramidal tract levy. Differential diagnosis (1) occlusive disease of medullary lesions common cause Only involving lateral medulla (ischemic softening) of this syndrome has emerged, as the scope of the lesions, Clinical manifestations vary, or even the performance of different syndrome. This levy is Avllis's syndrome and Babinski - Nageotte's a complex disease syndrome and sometimes difficult to identify. Avellis's syndrome is a disease damage to the medulla suspected nuclear and nucleus of the solitary tract and spinal cord Qiusuo performance ipsilateral soft palate, vocal cord paralysis and loss of sensation of throat, tongue after 1 / 3 of taste, since his head following the contralateral side body pain, temperature Cox obstacles. Suspected nuclear, the nucleus of the solitary tract, the trigeminal nerve cord, the hypoglossal nucleus, the body and the rope mesh structure damage, there Babinski - Nageotte's syndrome, the performance of the contralateral cerebellar ataxia partial body, sensory dysfunction, ipsilateral Horner's levy. This levy is Wallenberg's syndrome (artery occlusion of the lateral medulla caused lesions) with right pyramidal tract adjacent to the composite performance of the levy. (2) medullary syringomyelia (syringobulbia) empty often asymmetrical, signs and symptoms usually unilateral. Also extended the scope of the differences may not show complete Cestan - Chenais's syndrome. TB often associated with syringomyelia exist at the same time, multiple age of 20 ~ 30 years old, and occasionally occurred in childhood or later in adulthood. On the formation of hollow principle that the majority is a congenital dysplasia.