Uric acid nephropathy What are the clinical manifestations?

Uric acid nephropathy refers to as serum uric acid excretion have reduced or excessive formation of hyperuricemia induced renal damage. There is room for two renal damage: ① formation of urinary stones; ② uric acid induced renal parenchymal damage.
Both renal damage will be able to coexist, Its clinical manifestations as follows:
(1) Chronic hyperuricemia: more common in middle-aged and older men. With more than gout or gouty arthritis stone; early renal damage showed mild proteinuria, a small amount of red blood cell concentration and urinary dysfunction; late have hypertension, renal dysfunction, a small number of lead uremia. Visibility renal medulla frugal living within a radial needle-shaped crystal uric acid and renal interstitial chronic inflammatory changes.
(2) acute uric acid nephropathy: rapid onset, was particularly prevalent in the bone marrow proliferative diseases and cancer radiotherapy, and chemotherapy. Clinical manifestations of oliguria or anuria, and the rapid deterioration of renal function (ie, acute renal failure). This shows that a large number of urinary uric acid crystal and erythrocyte. If not promptly treated, the condition continues to worsen, the patient eventually died of renal failure.
(3) uric acid stones: Multi asymptomatic hematuria can happen, with or without renal colic, the translucent stone, abdominal plain film showed that no more often to go through IVP can be found. Urine Shicheng divided into uric acid or its salts.